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Forensic Medicine · Thanatology: post-mortem gastric digestion artefact
Sample itemAt autopsy of a man who died after a head injury, the fundus of the stomach and lower oesophagus are soft, brownish-black and thinned, with a perforation allowing gastric contents into the left pleural cavity. The edges show no hyperaemia, no inflammatory exudate and no haemorrhage. What is the correct interpretation?
Original practice item, not a recalled exam question. Nothing here is recorded.
The answer and the reasoning
D. Post-mortem digestion of the wall by gastric juice, an artefact
After death, gastric acid and pepsin can digest the stomach wall and adjacent oesophagus, especially when the stomach is full, producing softening (gastromalacia) and perforation. The absence of any vital reaction (no hyperaemia, exudate or haemorrhage) shows that the change is post-mortem. Ante-mortem lesions show inflammation or bleeding.
Option by option
A. Ante-mortem corrosive acid ingestion
Ante-mortem corrosives cause congestion, haemorrhage and a vital reaction, with a history of ingestion.
B. Perforated peptic ulcer with peritonitis
A perforated ulcer causes an inflamed, thickened edge and peritoneal inflammation.
C. Spontaneous rupture of the oesophagus (Boerhaave syndrome)
Boerhaave tears cause a linear tear with mediastinal inflammation, not a softened thinned wall.
D. Post-mortem digestion of the wall by gastric juice, an artefact (correct)
Correct. Lack of vital reaction with thin, brown-black softened wall indicates post-mortem digestion.
Source note: Original NextPrep sample item — pending expert review. This item is original practice material awaiting independent expert review, so confirm important facts in a standard textbook.
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