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Orthopaedics · Peripheral nerve injuries: Vaughan-Jackson syndrome
Sample itemA woman with long-standing rheumatoid arthritis suddenly cannot extend her little finger at the metacarpophalangeal joint, and over the next weeks the ring finger follows. The head of the ulna is prominent. What is the mechanism?
Original practice item, not a recalled exam question. Nothing here is recorded.
The answer and the reasoning
C. Attritional rupture of extensor tendons over a prominent ulnar head
In Vaughan-Jackson syndrome, synovitis and a dorsally subluxed ulnar head erode the extensor tendons, so they rupture one by one from the ulnar side, starting with extensor digiti minimi. A posterior interosseous nerve palsy would weaken all digits and the thumb together. Treatment is tendon transfer or grafting with ulnar head resection.
Option by option
A. Ulnar nerve compression at the cubital tunnel
Cubital tunnel compression causes ulnar intrinsic weakness and clawing, not loss of metacarpophalangeal extension.
B. Posterior interosseous nerve palsy
A posterior interosseous palsy would paralyse finger and thumb extension together, not one finger at a time.
C. Attritional rupture of extensor tendons over a prominent ulnar head (correct)
Correct. Sequential tendon rupture from the ulnar side over a prominent ulnar head is the classic pattern.
D. Stenosing tenosynovitis of the A1 pulley
A trigger finger gives painful catching in flexion, with no loss of active extension.
Source note: Original NextPrep sample item — pending expert review. This item is original practice material awaiting independent expert review, so confirm important facts in a standard textbook.
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